NaV1.5 in embryonic heart development
dc.contributor.author
dc.date.accessioned
2020-09-15T11:35:41Z
dc.date.available
2022-10-28T05:46:33Z
dc.date.issued
2020-10-01
dc.identifier.issn
1748-1708
dc.identifier.uri
dc.description.abstract
The voltage‐gated sodium (NaV) channel is essential for cardiomyocyte function. It is responsible for generating the rising phase of the cardiac action potential. The channel consists of a pore‐forming, α‐subunit, whose main cardiac isoform is NaV1.5, and one or two associated β‐subunits. Moreover, it is part of a macromolecular complex including a diverse set of interacting proteins, some of which are implicated in regulating its localization to specialized membrane domains of the cardiomyocyte sarcolemma
dc.format.mimetype
application/pdf
dc.language.iso
eng
dc.publisher
Wiley
dc.relation.isformatof
Versió postprint del document publicat a: https://doi.org/10.1111/apha.13542
dc.relation.ispartof
© Acta Physiologica, 2020, vol. 230, núm. 2, p. e13542
dc.relation.ispartofseries
Articles publicats (D-CM)
dc.rights
Tots els drets reservats
dc.source
Cortada Almar, Èric Vergés Aiguaviva, Marcel 2020 NaV1.5 in embryonic heart development Acta Physiologica 230 2 e13542
dc.subject
dc.title
NaV1.5 in embryonic heart development
dc.type
info:eu-repo/semantics/article
dc.rights.accessRights
info:eu-repo/semantics/openAccess
dc.embargo.terms
2021-10-01T00:00:00Z
dc.date.embargoEndDate
info:eu-repo/date/embargoEnd/2021-10-01
dc.type.version
info:eu-repo/semantics/acceptedVersion
dc.identifier.doi
dc.identifier.idgrec
031763
dc.type.peerreviewed
peer-reviewed
dc.identifier.eissn
1748-1716